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Canada-0-Uniforms 회사 디렉토리
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회사 뉴스 :
- Acinetobacter baumannii DacC influences cell shape, biofilm . . .
In summary, we conclude that dacC encodes a dual enzyme, possessing activities of DD-CPase and β-lactamase, which significantly affects the physiology of A baumannii in various ways whereas dacD encodes a strong DD-CPase and plays a role in cell morphology, though it exerts negligible impact on other physiological aspects like intrinsic
- The physiological role of Acinetobacter baumannii DacC is . . .
The physiological role of Acinetobacter baumannii DacC is exerted through influencing cell shape, biofilm formation, the fitness of survival, and manifesting DD-carboxypeptidase and beta-lactamase dual-enzyme activities
- Acinetobacter baumannii Biofilm Formation and Its Role in . . .
Acinetobacter baumannii is an emerging global antibiotic-resistant gram-negative bacteria that most typically causes biofilm-associated infections such as ventilator-associated pneumonia and catheter-related infection, both of which are resistant to antibiotic therapy
- Factors mediating Acinetobacter baumannii biofilm formation . . .
Among several identified OMPs, outer membrane protein A (ompA) is a well-characterized virulence factor owing to its diverse key roles in the survival and pathogenesis of A baumannii, including maintenance of cell membrane integrity, mediating drug resistance, modulation of host immune response, initiation of biofilm formation, invasion of
- A polyamine acetyltransferase regulates the motility and . . .
Expression of dpa increases in bacteria that form pellicle and adhere to eukaryotic cells as compared to planktonic bacterial cells, suggesting that cell motility is linked to the pool of
- Biofilm formation in Acinetobacter baumannii - PubMed
This review summarises current knowledge on A baumannii biofilm formation and its clinical significance, as well as the related genetic determinants and the regulation of this process
- Acinetobacter baumannii DacC influences cell shape, biofilm . . .
Surprisingly, the deletion of dacC resulted in a reduced growth rate, loss of rod-shaped morphology, reduction in biofilm-forming ability, and enhanced susceptibility towards β-lactams, whereas, the deletion of dacD had no such effect
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